Engineered protein that cuts Alzheimer's-linked brain deposits opens potential therapeutic avenue

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A KAIST-led research team reports that a gene-regulating protein can suppress amyloid-beta aggregation associated with Alzheimer’s disease. The protein also reduced toxicity in aggregates that had already formed, suggesting a possible therapeutic research avenue.

A KAIST-led research team has found that a protein involved in gene regulation can suppress the harmful aggregation of amyloid-beta, a peptide associated with Alzheimer's disease. The approach also reduced the toxicity of protein aggregates that had already formed.